I have a hypokalemia paralysis with normal level of thyroid I continue my medicine from one month but still my poytasium is low day by day and I take everyday potkular potassium syrup i want to cure permanently so give me best best option.

Hypokalemic periodic paralysis is a disorder that causes occasional episodes of muscle weakness and sometimes a lower than normal level of potassium in the blood. It is one of a group of genetic disorders that includes hyperkalemic periodic paralysis and thyrotoxic periodic paralysis. Causes hypokalemic periodic paralysis is congenital. This means it is present at birth. In most cases, it is passed down through families (inherited) as an autosomal dominant disorder. In other words only one parent needs to pass the gene related to this condition on to you in order for you to be affected. Occasionally, the condition may be the result of a genetic problem that is not inherited. Unlike other forms of periodic paralysis, persons with congenital hypokalemic periodic paralysis have normal thyroid function. But they have a very low blood level of potassium during episodes of weakness. This results from potassium moving from the blood into muscle cells in an abnormal way. Risk factors include having other family members with periodic paralysis. The risk is slightly higher in asian men who also have thyroid disorders. The most important treatment in severe hypokalemia is addressing the cause, such as improving the diet, treating diarrhea, or stopping an offending medication. Patients without a significant source of potassium loss and who show no symptoms of hypokalemia may not require treatment. Mild hypokalemia (>3.0 meq/l) may be treated with oral potassium chloride supplements (klor-con, sando-k, slow-k). As this is often part of a poor nutritional intake, potassium-containing foods may be recommended, such as leafy green vegetables, tomatoes, coconut water, citrus fruits, oranges, or bananas.[14] both dietary and pharmaceutical supplements are used for people taking diuretic medications. Severe hypokalemia (<3.0 meq/l) may require intravenous supplementation. Typically, a saline solution is used, with 20? 40 meq/l kcl per liter over 3? 4 hours. Giving iv potassium at faster rates (20? 25 meq/hr) may predispose to ventricular tachycardias and requires intensive monitoring. A generally safe rate is 10 meq/hr. Even in severe hypokalemia, oral supplementation is preferred given its safety profile. Sustained-release formulations should be avoided in acute settings. Difficult or resistant cases of hypokalemia may be amenable to a potassium-sparing diuretic, such as amiloride, triamterene, spironolactone, or eplerenone. Concomitant hypomagnesemia will inhibit potassium replacement, as magnesium is a cofactor for potassium uptake. when replacing potassium intravenously, infusion by a central line is encouraged to avoid the frequent occurrence of a burning sensation at the site of a peripheral infusion, or the rare occurrence of damage to the vein. When peripheral infusions are necessary, the burning can be reduced by diluting the potassium in larger amounts of fluid, or mixing 3 ml of 1% lidocaine to each 10 meq of kcl per 50 ml of fluid. For more doubts you can ask me privately. 

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